Physical Conditioning and Training
Physical Conditioning and Training
by Cameron Martz
Cardiovascular
Conditioning
Some experienced
divers believe that exercise will do little for their diving
since they already have a good breathing rate and efficient
water skills. This is a very limited
view of what their heart and lungs do for them. Achieving a high level
of cardiovascular fitness does a lot more than just improve
your gas consumption. It may also increase
the safety of your dives in several ways:
1.
Increased physical reserves for dealing with
problems.
2. Delayed/reduced panic response.
3. Increased rate of inert gas elimination.
4.
Reduced cost of free phase gas formation.
Increased Physical
Reserves:
This is a no-brainer.
The fitter you
are, the more physically demanding a task you can handle
successfully. You
can swim faster and farther. You can manage larger
amounts of equipment. Your heartrate and
respiration are lower, and gas consumption will increase less
for a given increase in workload. There are no more
obvious results of cardiovascular conditioning than
these.
Imagine being
able to upgrade your car from your current engine to one that
is more powerful and gets better gas
mileage. As your
muscle cells adapt to exercise, they increase their number of
mitochondria (the energy machines of the cell) and their
quantity of aerobic enzymes (the oxygen-utilizing chemicals).
With these
adaptations, muscle cells become stronger while becoming much
more efficient with the oxygen they receive. A fit diver will thus
be able to perform more work with each breath of gas, or use
less gas to perform the same amount of work as a less fit
diver. Not only
does this give you the capacity to do more during each dive,
this also increases the chance that you can solve any problems
that might arise.
The Panic
Response:
An important side
benefit of a reduction in heart rate and respiration relates
to the panic response. The human brain
responds to an increase in heart rate and respiration with an
increased emotional response, whether it is love, anger, or
panic. A feedback
loop forms in a tense situation when a diver senses danger,
then responds with an increase in physical activity. This causes the divers
heartrate and respiration to increase, which results in the
brain increasing its perception of danger, which then elevates
the divers heartrate and respiration, which then further
increases the brains perception of danger, and so on until the
diver can no longer perform the appropriate response. Thus, the fitter you
are, the further you will be from your panic threshold merely
because your heartrate and respiration are not as affected by
increasing physical demands.
Increased Rate of
Inert Gas Elimination:
The rate at which
inert gas is eliminated from the tissues of your body for a
given pressure gradient depends upon the solubility and
vascularity of the tissues and the efficiency of your lungs.
Fat tissue
off-gasses much more slowly than lean tissue, partly because
it holds a greater quantity of dissolved gasses than other
tissues. This
storage problem is further compounded by the tissues low
vascularity. Cardiovascular
training, when combined with a healthy diet, will result in an
increased ratio of lean tissue to fat tissue in an athletes
body. The body of
a fit diver will off-gas as a system faster than that of an
unfit diver.
Keep in mind
that a reduction in adipose tissue, or body fat, reduces the
amount of natural insulation a diver has. Thus, adequate
protection from the water becomes even more important for the
fit diver.
Cardiovascular
training also increases the efficiency of the lungs through
several mechanisms. As you overload your
cardiovascular system through exercise, you stimulate your
lungs to exchange carbon dioxide and oxygen, primarily, at a
much faster rate. Your body adapts by
increasing the vascularity of the lung tissue as well as
increasing the surface area of the lungs at the alveoli. Not only is a greater
quantity of blood present in the lungs of a fit diver, but the
vascular changes also allow a faster rate of gas diffusion for
each unit volume of blood. Fortunately, these
adaptations are not specific to oxygen or carbon dioxide- a
pressure gradient for any gas will result in an increased
transfer of that gas from blood to lungs. We will return to the
importance of these effects later.
Reduced Cost of Free
Phase Gas Formation:
Every dive has the
potential to produce bubbles, whether it be a thirty-minute
shallow reef dive or a world record setting deep cave
penetration. The
size and amount of bubbles formed depend primarily upon the
amount of dissolved gas and the rate of ascent. Mismatch the rate of
ascent for the amount of dissolved gas, and the bubbles set in
motion a series of problems, known to divers as Decompression
Sickness (DCS).
Contrary to
popular belief, bubbles are not the only cause of blockages in
the circulation. The arterial
capillaries are generally large enough to allow the passage of
many free phase bubbles. Rather, it is believed
to be the secondary effects of these bubbles that cause many
of the blockages, or emboli, associated with DCS.
The emboli
believed to be associated with DCS result from several
sources. The body
releases several types of chemicals in response to the
vascular insult resulting from bubble formation, and these
chemicals have been shown to reduce the blood supply to the
tissues, even without the presence of gas emboli. Additionally, certain
proteins involved in the bodys defense against illness may
adhere to the bubbles themselves, causing blockages and
decreasing the permeability of the bubbles. These proteins not only
increase the size of the bubbles, but they also increase the
time required to clear the bubbles out of the
bloodstream.
Divers must
always keep in mind that our lungs are our first defense
against the effects of breathing compressed gas. The diffusing capacity
of the lungs is much greater than needed at rest. This built-in safety
factor is what allows the lungs to act as a very effective
bubble filter in the event of free phase gas formation in the
bloodstream. The
alveoli are designed to trap both solid and gaseous emboli,
preventing them from traveling further through the
circulation. Gaseous emboli are
eliminated through diffusion, which as described above, is
improved through cardiovascular conditioning. Cardiovascular
conditioning further increases this safety factor by allowing
the lungs to trap a greater quantity of bubbles within their
increased surface area and vascularity.
Not all emboli
are filtered by the lungs, however. Small bubbles can pass
through the pulmonary circulation only to collect and form
emboli elsewhere in the body. Also, the accumulation
of proteins and platelets occur throughout the circulatory
system as a result of free phase gas formation. This is where the other
vascular effects of cardiovascular conditioning may become so
important.
The diameter of
blood vessels varies based upon a number of factors, including
vascular insult. However, cardiovascular
conditioning increases the maximum possible diameter of many
existing blood vessels. Thus, an embolus may
travel further downstream before becoming lodged. The further down the
circulation an embolus can pass, potentially fewer branches
will be blocked and less tissue will be affected.
Cardiovascular
conditioning increases collateral circulation, which means
that a given mass of tissue may have more pathways from which
to receive oxygen-rich blood. Thus, if an embolus
becomes lodged in one pathway, the tissues of a fit diver may
receive more blood than those of an unfit diver via other
pathways.
Cardiovascular
conditioning also increases the efficiency with which cells
utilize the oxygen they receive. In other words, tissues
of a fit diver require less oxygen to maintain their base
metabolic rate than those of an unfit diver. This has to do with an
increase in aerobic enzymes contained with in the cells, as
well as a few other structural changes to the cells. Thus, tissues of a fit
diver may better survive a reduction in blood supply compared
to those of an unfit diver.